❔ antipsihotiki - 3

Nisi edini, tudi avtorica jih jemlje. Kvetiapin ( seroquel/ kventaks/ kveluks itd)

Kakšne težave pa imaš, da jih jemlješ?

To bo morala avtorica - "mama"- povedati.
Za kaj pa so jih predpisali tebi?

Se en problemcek nastopi pri zdravljenju depresije / anksioznosti z antipsihotiki + antidepresivi.
Zdravnik bo povedal ( ce je pacient sploh toliko informiran, da ve, kaj jemlje)
da pri depresiji predpise Nizke doze Antipsihotikov

Niti ne.

ABILIFY 10 do 15 mg/dan za shizofrenijo, 2mg-15 mg / dan za depresijo kot dodatek.

The efficacy of ABILIFY as an adjunctive therapy for major depressive disorder was established within a dose range of 2 mg/day to 15 mg/day.
http://www.drugs.com/dosage/abilify.html#Section_2.3

Dodaten problem nastopi, ker se tako Antidepresiv kot Antipsihotik presnavljata v jetrih in pri tem uporabljata iste encime.
(CYP3A4 in CYP2D6)
Antidepresiv ( fluoexetine, paroxetine, sertraline) tako blokira razgradnjo antipsihotika ( ker je inhibitor encima)
in s tem poveca njegovo kolicino v plazmi, in seveda njegovo ucinkovitost za tudi vec kot 100%.

Torej pacienti z depresijo, s kombinacijo AD + AP ne jemljejo nizkih doz, pac pa se mocnejse, kot bi
jih jemali pri shizofreniji.

Glede na moje izkušnje s psihiatri bi rekla, da ne samo da ne povedo tega, ampak niti približno ne vedo. Res, koliko nesposobnih ljudi... hb ga sama še nisem rabila, a tudi če bi ga, sploh ne vem, kaj bi. Kakšno leto iskala enega, ki je sposoben, profesionalen, zainteresiran (tako zame kot za nenehno izobraževanje)... misija nemogoče.

kaj pa meniš o šentjanževki proti depresiji (sori, če si že pisala o tem)

Sentjanzevka - hypericum perforatum - naj bi pomagala pri zmerni do srednje tezki depresiji.( karkoli ze to je...)
Studij na to temo je malo.
Ucinkovine hyperforin in hypericin naj bi delovale - neselektivno - na ponoven privzem "mnogih" ( ?) nevrotransmiterjev, predpostavljajo, da naj bi
( na se nepoznan nacin) delovale na sigma receptorje.
Dejstvo je, da dvigne razpolozenje, uporablja pa se ze od pamtiveka.

http://www.nature.com/articles/srep05632

Raziskave so pokazale enako ucinkovitost sentjanzevke in fluoxetina / sertralina/ imipramina.
Pacienti so, kot je bilo tudi pricakovano, lazje prenasali sentjanzevko. ( viz. stranski ucinki)

"Hypericum has been compared to leading antidepressant medications. In a randomized, controlled, double-blinded trial,
70 patients suffering from mild to moderate depression received 1 tablet of either hypericum extract or fluoxetine twice a day for 6 weeks. As evaluated by the 17-item Hamilton Rating Scale forDepression (HAMD), the von Zerssen depression scale (DS), and
patients’ response, there were significant decreases (P<0.001) in symptoms in the hypericum group (50%) and in the fluoxetine
group (58%) on their HAMD score. The hypericum extract achieved 83% of the efficacy of fluoxetine on the HAMD and
78% on the DS. Assessments by physicians and patients indicated considerable improvement with no between-treatment
differences.
25 The authors concluded that the hypericum tested in this study was therapeutically equivalent to fluoxetine and
that it is a reasonable alternative to synthetic antidepressants.

Hypericum extract has similarly been tested and proven at least as effective as sertraline in the treatment of mild to
moderate depression in a small group of outpatients.
26 Efficacy and tolerability of hypericum extract was also compared with imipramine and was found equivalent to the drug in treating
mild to moderate depression. In addition, as expected, patients tolerated the hypericum better.
27 Overall, the literature supports the use of hypericum extract for patients with mild to moderate
depression.

Hypericum

razmišljam o nečem, ker se res grozno počutim, ampak na ad pa ne bi šla. mislim, da bom poskusila tole.

Pa Omega 3 v precejsnjih kolicinah.
I`m not joking.
Tudi ce ne bo delovalo pozitivno, bodo edini stranski ucinki lepsa koza, nohti in lasje 🙂

bom tudi to poskusila... res je, škodit ne more, je lahko samo plus.

In ko se bos pocutila boljse, se malo aerobicne vadbe.

Exercise-for-depression

Dejstvo je, da je veliko na tem "dihat in se premigat".
Vendar, ko se clovek pocuti grozno, preprosto ne more, in mu podobni nasveti lahko zvenijo skoraj kot zalitev-
ces, kaj pa TI ves, koliko energije porabim samo za to, da se sploh premikam naokoli?

Dejansko je tako ( ce uporabim avtomobilsko prispodobo) kot da bi
vozil z zategnjeno rocno zavoro.
Neizmerna utrujenost je ena stvar, potem pa mogoce se obcutek krivde in samoobtozevanje, ker ne naredi nic zase.

Vendar, ko se pocuti boljse, je dobro "vtihotapiti" malo aerobicne vadbe. In potem zmeraj vec, in pocutje bo cedalje boljse 🙂

2 tedna nazaj sem se komaj iz postelje premaknila. če si nisem naredila budilke, sem spala 13 - 14 ur. take cikle mam, ko je že malo boljše, potem pa spet nazaj. in imaš čisto prav, ker se dejansko po kakšnem sprehodu boljše počutim, ampak bi morala bit bolj vztrajna, res vsak dan.

Mogoce je tudi izgorelost v igri, zato si poskusi omogociti, da obcasno res spis 14 ur, ce to telo potrebuje, ne da bi se pocutila krivo.
Spanec pa je boljsi in bolj zaleze, ce se predihas in telesno utrudis.
Ena od zoprnih stvari pri depresiji je, da clovek slabo zaznava signale lastnega telesa - tezko obcuti pravo lakoto, pravo utrujenost, pravo zaspanost.
Ampak, to se popravi.

glede na to, da sem v težkem položaju že 3 leta, da me je dohitelo - in povozilo.
ne vem, dobro se počutim, ko spim. in če se že predramim, mi ni za vstati (če ni res treba). pač tako je, ko nimaš kakega posebnega veselja za dan, ki je pred tabo. je pa res, da sem že od nekdaj potrebovala več spanca kot drugi, optimalno mi je bilo 9 ur 🙂
najprej sem mislila, da je tole samo stopnjevanje moje garfieldovske narave... potem sem pa izgubila veselje do dveh hobijev, tako sem začela razmišljati, da je resnejša zadeva. spremembe v počutju so bile dolgotrajne in postopne, tako da sama težje ocenim, kdaj je stanje postalo bistveno slabše, vem pa da nikoli nisem bila tako pesimistična.
hvala za spodbudne besede in vse nasvete, saphire

You are welcome 🙂
V bistvu ni vazno, kdaj, ker zmeraj ukrepamo iz tocke nic, iz Tukaj in Zdaj.
Pri depresiji ( ali pac depresivnem pocutju, mizernem pocutju, zhblj pocutju ) se je dobro izogibati dvem stvarem - tuhtanju in obcutkom krivde.
Tuhtamo in razglabljamo, zato ker imamo obcutek, da smo se nenadoma znasli v "breznu", vortexu, v nerazlozljivih tezavah. In bi radi to razlozili, logicno razclenili in se z mocjo razuma potegnili ven. Samo to ne gre, in potem se zaciklamo.
Obcutki krivde so navadno predimenzionirani, vcasih do absurdne mere, pa tudi ce niso, so kontraproduktivni.
Gre za napacno sprejet signal.
Tudi, ce je treba kaj popraviti, bomo to lahko naredili sele, ko se pocutimo boljse, ne pa medtem, ko smo v vortexu.
Lahko se uporabi pravzaprav otrocji trik, in sicer alokacija casa.
Ce se zalotimo pri tuhtanju ali samo-okrivljanju, si recemo - to bom pocela v petek od petih do pol sedmih, NE PA ZDAJ. Itd.
Vcasih deluje 🙂
Lahko deluje tudi prizemljanje ( grounding) tapkanje, - eft, akupresura proti depresiji.
Pa seveda smeh in humor, kadar le lahko. Smeh, globok smeh iz trebusne prepone dela cudeze, believe you me 🙂)

Prednosti mascobnih kislin Omega3


Fighting Depression and Improving Cognition with Omega-3 Fatty Acids

"Brain tissue is rich in omega-3 fatty acids, which are of vital importance within cell membranes and in connections between nerves
Low dietary intake of beneficial omega-3 fatty acids, such as eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA), is linked to depressed mood, hostility, and impulsive behavior.
High intake of EPA and DHA is associated with increased gray matter volume in brain regions controlling depression and mood.
Low levels of omega-3 fatty acids are linked to poor performance on cognitive and memory tests, and more rapid cognitive decline with aging.13,14 High intake of omega-3 fatty acids during pregnancy is associated with better neurodevelopmental outcomes later in childhood.19,20
In controlled clinical studies, depressed patients randomly assigned to receive omega-3 fatty acids have demonstrated greater improvement compared with those assigned to placebo."

Odpri sliko ➜

poskusila. res imaš naštudirano tole... upam da ne zaradi lastnih izkušenj..? če pa, si pa tudi očitno uspešno prebrodila.

Moja osebna izkusnja z AD je bila v 1998 - 6 mesecev - in to s famoznim Prozacom 😆 ( fluoxetine)
Ne spomnim se nekih tezkih stranskih ucinkov, razen glavobola zjutraj in pa nenadne enormne kolicine zivcne energije, ki sem jo potem skusala "pokuriti" z res dolgimi sprehodi in z intenzivnim ciscenjem - se nikoli se niso ploscice in okna tako svetila 😛
Prenehala sem jih jemati naenkrat - takrat se ni dosti vedelo ali govorilo o "tapering" / postopnem zmanjsevanju doze, ki ga zdaj vsekakor priporocajo. Tudi odtegnitvenih ucinkov se ne spomnim, (mogoce so bili kaksni gripozni obcutki)- tako, da sem imela sreco.

Dejstvo je, da AD-ji LAHKO pomagajo, se posebej pri hudi depresiji, ko clovek lahko samo se zdi in se mu po glavi podijo samo se crne misli.
( jaz nisem imela tako skrajne.)
Ad-ji zalezejo, "primejo" pri vsaj 60% ljudi ( stevilke se sicer divje razlikujejo)
Bistvo pa je, da delujejo kratkorocno - telo vzpostavi homeostaticno ravnotezje in se preneha odzivati na umetno dvignjen serotonin

The mechanism of Tolerance in Antidepressant Action

In bistveno tudi, kaj clovek naredi,kako izkoristi obdobje, ko AD ji delujejo pozitivno.
Lahko, da se depresija v 6 mesecih poleze sama od sebe.
Lahko, da se clovek loti mehanizmov, ki mu pomagajo razvozlati tezave, ki so privedli do depresivnega pocutja, ali pa
mehanizmov, ki mu pomagajo ziveti s temi tegobami

Ce pa se zanasa samo in edino na tabletke, potem lahko nastopi problem. Ko prenehajo delovati, zvisajo dozo, potem poskusijo z drugim AD, potem z dvema hkrati, potem z AD in AP hkrati.... In nenadoma ima clovek vec problemov ( od zdravil), kot osnovnih problemov.
Tisto, kar jaz menim, da je narobe, je vzdrzevalna terapija - jemanje AD dolgorocno - in pa jemanje AD za nedepresivna stanja.

Pa za tisto gospo, ki je sprasevala o sentjanzevki -
dobro je, da bi si dala narediti natancno hormonsko sliko, preiskave za vitamine ( posebej D), in za krvni sladkor.

Naslednja stanja namrec skoraj popolno oponasajo depresivne simptome :

Hipotiroza ( hipertiroza pa oponasa anksioznost)
depression-bipolar-disorder-and-hypothyroidism/
Pomanjkanje Vitamina D
Low Vitamin D levels predict depression
Prenizek krvni sladkor in insulinska rezistenca
Insulin resistence and depressive symptoms

Tudi alergija na doloceno hrano, dehidracija
http://today.uconn.edu/2012/02/even-mild-dehydration-can-alter-mood/
in prevec kofeina lahko vplivajo na razpolozenje v tem smislu.

res! si svojo zgodbo kje objavila? - bi verjetno lahko prav prišla ogromno ljudem.

Do zdaj ne 😛
No, pa saj moja izkusnja ni bila nic posebnega. L.98 me je pac "doletela" depresija, najverjetneje reaktivne narave - nalepke niso niti vazne, danes bi mi mogoce nalepili "sindrom pretiranega zalovanja". Ko sem prisla do tocke, kjer se mi je zdelo popolnoma nemogoce vstati iz postelje in se obleci, kaj sele iti ven, sem se prisilila in sla do zdravnika. Jemala sem potem ta famozni Prozac, in ,kot receno, nobenih pretresljivih ucinkov- razen ta zivcna energija / nervous energy/ zaradi katere se mi je vcasih zdelo, da bom skocila iz koze, ce se ne bom premikala. Tako sem pac glancala - tla, kopalnico, pohistvo- vse kar se je glancat dalo... 🙂
Moje misli in obcutki se niso kaj dosti spremenili.
Vsak dan sem se prisilila, da sem sla v bazen odplavat vsaj 20 dolzin, kar je bilo vec, je bil pa bonus.Drugace nisem pocela nic posebnega.
Oziroma ne - vsake 2 tedna sem se sla pogovarjat z dvema terapevtoma - eden je bil trainee ( vajenec). Tako on, kot njegova mentorica sta bila zelo v redu, zelo fajn cloveka. Nismo se sli nobene poglobljene psihoanalize, pravzaprav smo si vsaj polovico seanse pripovedovali vice. No, to bolj kasneje, na zacetku mi ni bilo prevec do hecanja. Ceprav rada zabavam ljudi, tudi kadar se ne pocutim prevec dobro.
No, po kakih 2 do 3 mesecih sem se zacela pocutiti znosno ( da recimo nisem vec izbruhnila v jok ob vsakem "sprozilcu"), po 5 mesecih pa kar OK in sem rekla, da bi pa mogoce nehala s tem prozacom. Svetovali so mi, naj se pocakam par tednov," da bo ziher", in tako je tudi bilo.
Biila sem se vseeno potrta in zalostna, vendar je bilo to popolnoma obvladljivo.
Torej nimam nobene dramaticne zgodbe ( npr. "My Prozac Hell" 😛 )
Depresija je minila, vendar ne morem reci, da ne bi tudi brez tablet.Na nek nacin bi minila.
Mogoce bi trajala dlje. Kdo ve...
Kasneje sem slisala se mnogo zgodb, veliko bolj zalostnih in dramaticnih, tako da
imam vecino izkusenj iz "druge roke".

Tablete, terapevti......... sem slišala........ itak, da postaneš totalka bolan!
Pol pa še ta čvek, kjer te do konca zdriblajo - ene ste res samouničevalke!

Ni nujno, da postanes totalno bolan. Zna se zgoditi, da postanes malo bolj zdrav, ali pa nekje isti. 🙂

da si bila samo ena izmed mnogih, ki jo je to doletelo, samo si pa ena redkih, ki se je nato tudi poglobila v to. in, vsaj pri meni, so kakšni alternativni pogledi vedno dobrodošli. seveda je na koncu vse odvisno od posameznika, od njegove osebnosti in oblike depresije, ampak ne bi bilo slabo, če bi tole prebrali. jaz sem kr iskala po forumih veliko, pa je redko kaj uporabnega, kakšni konretni nasveti. moti me tudi prevladajoče mnenje psihiatrov, da ni nič narobe s tem, če ješ ad celo življenje. dobesedno ne da se jim ukvarjat s tabo in na ta način imajo najmanj dela, samo recepte obnavljajo. da ne omenjam tega, da ima znanka zdaj že kar nekaj časa precej slabe izvide jetrnih testov (in ker ni spremenila česa drugega, razen ad je začela jest 5 let nazaj, je prepričana, da so le-ti krivi)... načeloma se vsi zavedamo škodljivosti tablet, ampak ko te tako doleti se pa res začneš spraševati, a je bilo treba... no in zdaj ji je psihiatrinja zgolj zamenjala vrsto ad. o odvajanju od njih nista govorili, čeprav se zdaj počuti zelo dobro (ona ji je rekla, kar jejte jih zdaj še nekaj let pa bomo videli, a veste, eni jih jedo celo življenje). 🙁

"kar jejte jih zdaj še nekaj let pa bomo videli, a veste, eni jih jedo celo življenje" - no, tole je pa obupna izjava. Nekaj let??
In fraza, - eni jih jejo celo zivljenje - naj bi ji bila v nekako tolazbo?
Psihiatrinja sama, ce bi bila v njeni kozi bi jih jedla "celo zivljenje"? Nekako dvomim v to.

Niso problem samo jetrni testi in ostali mozni "stranski" ucinki.
Cedalje vec je podatkov, ki kazejo, da pri dolgotrajnem jemanju
antidepresivi ne samo izgubijo ucinek, pac pa zacnejo ucinkovati obratno-
torej depresivno / disforija, povzrocena z antidepresivi/
Poleg tega, ko se enkrat izgubi pozitivni odziv, nadaljni tretman, povecevanje doze, dodajanje drugih AD, lahko vodi
do depresije, odporne proti zdravljenju in do kronicne depresije.
Tako da, paradoksalno vendar resnicno, dolgotrajno bombardiranje mozganov
z antidepresivi, lahko povzroci osiromasenje serotonina
in s tem kronicno / neodzivno depresijo.


"In the early 1990s, only about 10% to 15% of patients with major depressive illness had treatment-resistant depression (and thus were chronically ill.) In 2006, researchers reported that nearly 40% of patients were now treatment-resistant. In a period when use of SSRI antidepressants exploded, refractory depression went on the march.

This condition, El-Mallakh writes, often develops in people who had a good initial response to an antidepressant, and then continue taking the drug. However, up to 80% of patients maintained on an antidepressant suffer a recurrence of symptoms, and once that “initial treatment response is lost,” continued efforts to treat the relapsed patient with antidepressants frequently results in “poor response and the rise of treatment-resistant depression.” Ultimately, this process—the continual prescribing of antidepressants to someone who has become treatment resistant—may "make the chronic depression permanent.”

In his discussion, El-Mallakh notes that people without any history of depression who are prescribed an antidepressant for other reasons—anxiety, panic disorder, or because they are serving as “normal controls” in a study—may become depressed, with that depression at times persisting for a fairly long period of time after the antidepressant is withdrawn. The reason that antidepressants may have a “prodepressant effect,” El-Mallakh writes, is that “continued drug treatment may induce processes that are the opposite of what the medication originally produced.” This is the “oppositional tolerance” that Fava has written about, and this process may “cause a worsening of the illness, continue for a period of time after discontinuation of the medication, and may not be reversible.”

https://now-antidepressant-induced-chronic-depression-has-name-tardive-dysphoria

With antidepressants, the problem may be that patients, because of the “oppositional tolerance” process, end up with a depleted serotonergic system. The postsynaptic neurons end up with a reduced density of receptors for serotonin; in rat studies (link is external), long-term treatment with an SSRI led to markedly reduced levels of serotonin in "nine areas of the brain." El-Mallakh, in his paper, details several other ways that exposure to an SSRI may deplete serotonergic function, and notes that in experiments with young animals, such impairments are "associated with increased depressive and anxious behaviors."

In conclusion, El-Mallakh writes that "a chronic and treatment-resistant depressive state is proposed to occur in individuals who are exposed to potent antagonists of serotonin reuptake pumps [i.e. SSRIs] for prolonged periods. Due to the delay in the onset of this chronic depressive state, it is labeled tardive dysphoria. Tardive dysphoria manifests as a chronic dysphoric state that is initially transiently relieved by -- but ultimately becomes unresponsive -- to antidepressant medication. Serotonergic antidepressants may be of particular importance in the development of tardive dysphoria."

Res je zelo odvisno od posameznika, in ne samo od osebnosti, pac pa celotnega organizma , kako se bo odzval na ADje.
Bioloska psihiatrija trenutno raziskuje "Biotipe", ki naj bi dolocali pojavnost razlicnih motenj in tudi odzive na zdravila.
( glede tega sem kar skepticna. Morajo se oprijeti necesa drugega, ker se jim grad iz peska - teorija o kemicnem neravnotezju kot vzroku - cedalje bolj podira.)
Znano pa je , da imamo precej razlik v metabolizmu in zato razlicen odziv na zdravila, tudi psihiatricna.

Jetrna bolezen in jetrne poskodbe pri antidepresivih so pogosto podcenjene ( zakaj me to ne preseneca?) in nediagnosticirane.

Vsekakor je dobro da pogleda, ( pogugla "drug interaction") ce uziva se kaksna druga zdravila, tudi zeliscna in prehrambene dodatke.


Antidepressant Induced Liver Injury Underestimated
Life-threatening or severe drug-induced liver injury has been reported for some antidepressants, including MAO inhibitors, tricyclic/tetracyclic antidepressants, venlafaxine, duloxetine, sertraline, bupropion, nefazodone, trazodone, and agomelatine, Dr. Perlemuter and colleagues report.

Although no dose-response relationship has been clearly demonstrated, it is best to stick to the minimum effective dosages of antidepressants to reduce the risk for liver injury, they advise.

Behaviorism and Mental Health
An alternative perspective on psychiatry's so-called "mental disorders" | PHILIP HICKEY, PH.D.
More on the Chemical Imbalance Theory
by PHIL HICKEY on NOVEMBER 2, 2015

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On October 23, 2015, Jeffrey Lacasse, PhD, and Jonathan Leo, PhD, published an interesting article on Florida State University’s DigiNole Commons. The title is Antidepressants and the Chemical Imbalance Theory of Depression: A Reflection and Update on the Discourse. Dr. Lacasse is assistant professor in the College of Social Work at Florida State University; Dr. Leo is Chair of Anatomy and Professor of Neuroanatomy at Lincoln Memorial University. The article was originally published in the Behavior Therapist in the October 2015 issue, pages 206-213.

The article provides a concise overview of the chemical imbalance theory from its inception, through its vigorous promotion by pharma-psychiatry, to its present reduced, but not quite dead, state.

Here are some quotes from the article, interspersed with my comments:

“In the early 2000s, the serotonin metaphor of depression was widely advertised by the makers of antidepressants, including advertisements for citalopram, escitalopram, fluoxetine, paroxetine, and sertraline…In particular, Zoloft(sertraline) advertisements featuring the miserable ovoid creature were unavoidable in U.S. television and magazines. An on-line repository of direct-to-consumer advertisements for psychiatric drugs lists many from 1997–2007 referring to a chemical imbalance, across many drugs and diagnostic categories (Hansen, 2015a, 2015b).”

The Hansen references mentioned in the above quote are worth examining. Ben Hansen is the well-known psychopharmacological savant Dr. Bonkers. The Bonkers Institute is always worth a visit. The links for the above quote are 2015a, and 2015b.

. . . . . . . . . . . . . . . .

“Since chemical imbalance is often presented as a rationale for taking SSRIs, some such patients now understandably feel lied to by their clinicians. Levine (2014) calls this ‘Psychiatry’s Manufacture of Consent.'”

“… in a rare controlled experiment on this topic, one group of depressed students were told they had a confirmed serotonin imbalance underlying their depression, while a control group was not (Kemp, Lickel, & Deacon, 2014). The group who was told they had abnormal serotonin levels found medication more credible than psychotherapy and expected it to be more effective. They also had more pessimism about their prognosis and a lower perceived ability to regulate negative mood states, yet experienced no reduction in self-blame. These results suggest that the chemical imbalance explanation may indeed be helpful in persuading patients to take medication but that this is likely accompanied by undesirable effects.” [Emphasis added]

The Kemp, Lickel & Deacon (2014) article is, in my view, one of the most important pieces of research in this field. It provides clear evidence that falsely informing people that they have a brain abnormality is disempowering and damaging. The article can be accessed here. The truly compelling aspect of this matter is that such a piece of research needed to be done at all. Isn’t it obvious that lying to people in this way would be disempowering and destructive? Would any legitimate medical specialty routinely operate in this way?

. . . . . . . . . . . . . . . .

“Perhaps the most interesting part about both of these NPR pieces [that were referred to earlier in the article] is that neither reporter questioned the experts about the ethics of telling a falsehood to patients because you think it is good for them.”

“It is easy to imagine that a single prominent academic psychiatrist, authoring an Op-Ed in The New York Times, could have set the record straight on serotonin imbalance decades ago. Yet, to our knowledge, no one did so.”

If psychiatry were anything other than a branch of medicine (and I realize that’s debatable), it would have been mauled to destruction by the mainstream media long ago. But the media and the general public have a great respect for medicine, and psychiatry has been afforded an undeserved share of this respect. But, as I’ve mentioned in earlier posts, the mainstream media are beginning to see through the façade, and are finally reporting on the “diagnostic” proliferation, the false claims, and the destructive treatments.

. . . . . . . . . . . . . . . .

“When our physicians are educating us, we prefer they not tell us any lies, white or otherwise. Unfortunately, characterizing the chemical imbalance metaphor as a ‘little white lie’ communicates a paternalistic, hierarchical approach that sounds suspiciously like the days of medicine that we thought we had left behind. It’s a ‘little white lie’ if you’re a psychiatrist; if you’re a confused, vulnerable depressed person who agrees to take an SSRI after hearing it, you might not consider it so little. After all, if your trusted physician tells you that you have a chemical imbalance in your brain that can be corrected with medication, not doing so sounds foolish, if not scary (Lacasse, 2005). How many patients with reservations about SSRIs have agreed to take medication after being told this ‘little white lie’?”

The “little white lie” is, of course, a reference to the 2014 article by the very eminent and influential psychiatrist Ronald Pies, MD. In that article, Dr. Pies characterizes the chemical imbalance theory as “…this little white lie…”

Dr. Pies has also insisted – arguably delusionally – that psychiatry never promoted the chemical imbalance theory of mental illness. In a 2011 article he wrote:

“In truth, the ‘chemical imbalance’ notion was always a kind of urban legend – never a theory seriously propounded by well-informed psychiatrists.”

In the article in hand, Drs. Lacasse and Leo provide clear and abundant evidence to the contrary. They also, incidentally, provide a summary of Dr. Pies’ past financial relationships with pharmaceutical companies. Apparently the eminent doctor has received funding from Glaxo Smith Kline, Abbot Laboratories, and Janssen Pharmaceutica. He has also consulted for Apothe Com, a medical communications agency that assists pharma in the commercialization and promotion of new drugs.

“Pies blames the drug companies for running misleading advertisements about chemical imbalance, belatedly admits he should have said something sooner, but fails to mention that he was paid to help them promote their products at the time the advertisements were running.”

“We previously argued that the propagation of misleading advertising ‘is only possible in the absence of vigorous government regulation . . . or outcry from professional associations’…That outcry never came, and these issues weren’t addressed publicly until the patents for most blockbuster SSRIs had expired, and Big Pharma moved onto mood stabilizers and atypical antipsychotics. While we are hesitant to overemphasize conflicts-of-interest as an explanation for what has occurred, we can’t help but notice that the silence of psychiatry regarding chemical imbalance only ended when the profits had been extracted from the SSRI marketplace.”

Now that’s an interesting coincidence!

“Many mental health clients find it unacceptable, and perhaps a violation of ethical informed consent, for clinicians to give patients metaphorical explanations for their mental health problems and promote them as scientific truth.”

The chemical imbalance hoax, which was diligently and self-servingly promoted by pharma-psychiatry for decades, is perhaps the most destructive and far-reaching scandal of the modern era. As a theory it was refuted almost from its inception, but was nevertheless promoted by psychiatrists and by massive advertizing campaigns, and served to increase sales of psychiatric drugs in every corner of the globe. There is no way to calculate the number of lives that have been lost, or severely compromised, as a result of this activity.

Now, anti-psychiatry groups are exposing the truth, and pharma-psychiatry are quietly altering their message. But there have been no apologies; no congressional hearings; no indictments; no CEO’s fired; no psychiatrists censured. Just business as usual, as the pharma-psychiatry leaders prepare their next “great breakthrough” message.

Odpri sliko ➜

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http://www.behaviorismandmentalhealth.com/2015/11/02/more-on-the-chemical-imbalance-theory

Premagajte depresijo brez zdravil
6. FEBRUAR 2009
Če vas muči depresija, a bi radi pred uporabo antidepresivov preizkusili nekaj naravnih metod za njeno zdravljenje, smo za vas pripravili nekaj naravnih zdravil, ki dokazano dobrodejno vplivajo na naše počutje.

Vedenjska kognitivna terapija

Vedenjska kognitivna terapija ljudem pomaga spremeniti način razmišljanja. V nasprotju s tradicionalnimi pristopi se omenjena terapija osredotoča na ‘tukaj in sedaj’ – na težave, ki nas mučijo v določenem trenutku. Številne klinične raziskave po vsem svetu vedno znova dokazujejo, da je terapija ravno tako učinkovita kot antidepresivna zdravila. Po 20 terapijah se stanje bistveno izboljša kar 75 odstotkom pacientov.


Šentjanževka

Šentjanževka je splošno znana po svojih antidepresivnih učinkih. No voljo je v obliki tablet, kapsul in celo tekočine. Raziskave kažejo, da deluje antidepresivno, saj dvigne raven serotonina, norepinefrina in dopamina. Njeno učinkovitost so preiskovale številne študije in sklenile, da šentjanževka odlično deluje pri lažji do srednji depresiji in je vsaj tako učinkovit kot paroksetin (pri nas v zdravilu Seroxat).

S-Adenosilmetionin (SAMe)

SAMe je derivat naravne aminokisline, ki se nahaja v vseh celicah. Igra pomembno vlogo pri mnogih bioloških reakcijah, saj svojo metilno skupino prenaša v DNK, beljakovine, fosfolipide in biogenske amine. Več znanstvenih raziskav je pokazalo, da učinkovito zdravi depresije.



Svetlobna terapija

Že leta se svetlobna terapija uporablja za zdravljenje depresij, ki jih povzročajo kratki in temačni zimski dnevi. Pomanjkanje sončne svetlobe namreč vodi do izločanja hormona melatonina, ki povzroča slabo razpoloženje. Svetlobna terapija pomaga uravnavati telesu lastno ‘uro’ na enak način, kot to počne sonce. Čeprav svetlobna terapija običajno pomaga pri zdravljenju sezonskih težav, pa pozitivno vpliva tudi na splošno depresijo.

Vadba

Telesna aktivnost je zagotovo eden najučinkovitejših načinov za preprečevanje depresij, saj ta poviša raven serotonina v možganih, kar vodi do izboljšanja počutja. Učinek vadbe na počutje lahko brez težav primerjamo z učinki močnih antidepresivov kot je Sertralin.



Masaža

Eden najboljših stranskih učinkov masaže je ta, da izboljša naše razpoloženje in počutje. Pri masaži v naših možganih prihaja do kemičnih sprememb, zaradi katerih smo sproščeni in pomirjeni. Obenem se zmanjša nivo stresnih hormonov, kot je kortizol, nivo katerega se v povprečju zniža za kar 30 odstotkov. Obenem masaža zviša nivo serotonina in dopamina, ki lajšata depresijo.

5-Hidroksitriptofan (5-HTP)

5-Hidroksitriptofan (5-HTP) in triptofan prav tako spadata med naravne alternative antidepresivom. Ko naše telo prične proizvajati serotonin, najprej namreč naredi 5-HTP.

http://lifestyle.enaa.com/zdravje-in-prosti-cas/premagajte-depresijo-brez-zdravil.html

kaj je svetlobna terapija?

Svetlobna terapija


Fotobiomodulacija ali svetlobna terapija je učinkovit, ne invaziven in cenovno ugoden fizikalen pristop k zdravljenju. Zdravi mišično-skeletne poškodbe, poškodbe in bolezni živčevja, kožne bolezni in pospešuje celjenje akutnih in kroničnih ran. Predstavlja učinkovito podporo zdravljenju in blaženju bolečin.

Priročnik Svetlobna terapija


VIDEO »

DELOVANJE RDEČE IN NIR SVETLOBE

Fotobiomodulacija je uravnavanje celičnih procesov s pomočjo natančno določenih svetlobnih režimov. V terapevtske namene se uporablja predvsem rdeča svetloba in svetloba blizu infra-rdečega dela spektra (Near Infra Red − NIR). Rdeče in NIR LED-diode emitirajo svetlobo v širokopasovnem območju svetlobnega spektra (600-1000nm) v frekvenčnih pasovih širine 20 - 25 nm (možne so tudi posebne izvedbe s širšim spektrom). Za pokritje večjih pasov hkrati (npr. en pas v rdečem območju in en pas v NIR območju) je potrebno uporabiti več različnih diod ali eno diodo, ki oddaja pri več valovnih dolžinah hkrati.
Glavni razlog za uporabo svetlobnih virov, ki oddajajo svetlobo v rdečem spektru je, da hemoglobin te svetlobe ne absorbira in ta nemoteno prodira globoko v tkivo. Ta del spektra nima škodljivih učinkov z upoštevanjem omejitev svetlobnih režimov. Svetlobi v temu območju pravimo strokovno »FOTOTERAPEVTSKO OKNO«. Svetloba, ki sveti v fototerapevtskem oknu, se torej absorbira v naše celice, kar privede do odziva tkiva in terapevtskih učinkov.

DELOVANJE NA CELIČNEM NIVOJU

Mnogi fiziološki procesi so odvisni od svetlobnega ciklusa. V celicah so fotoreceptorske molekule, imenovane citokrom c oksidaze, ki so encimi dihalne verige v mitohondrijih. Mitohondriji so celični organeli, ki skrbijo za produkcijo celične energije. Citokrom c oksidaza absorbira svetlobo v fototerapevtskem oknu (NIR- in R-spekter), kar iniciira zaporedje redoks reakcij na notranji membrani mitohondrija. Fotobiološki odgovor teh primarnih mehanizmov se odraža v transdukciji fotosignala do sekundarnih mehanizmov, ki zagotavljajo vrsto kliničnih učinkov: celjenje ran, izboljšanje prekrvitve in limfnega pretoka, regeneracijo in imunomodulacijo.
S fotobiomodulacijo omogočimo znatno podporo organizmu pri vzdrževanju telesne homeostaze.

FOTOBIOMODULACIJA UČINKI

Mnoge znanstvene raziskave, med njimi tudi klinične, potrjujejo vpliv svetlobe na zdravljenje različnih bolezni in poškodb.
Svetloba pripomore k ohranjanju ali ponovnemu vzpostavljanju ravnovesja v organizmu že na celični ravni, kjer:
Deluje antioksidativno in pripomore k zaščiti celic pred prostimi radikali.
Vpliva na proteine, ki varujejo celice pred degenerativnimi procesi.
Poveča osmotsko odpornost eritrocitov in s tem preprečuje hemolizo.
Vpliva na rastne dejavnike živčevja in mikrožilja ter mišičnega in vezivnega tkiva. Uravnava vnetje, imunski odziv in cirkadiane ritme. Podpira obnovo tkiv.
Spodbuja lokalno prekrvitev.
Podpira limfni sistem.
Svetloba vpliva na mehanizme bolezenskega dogajanja in tako bistveno pospeši okrevanje. Obsevano mesto je bolje prekrvljeno, izboljša se presnova celic in spodbudi regenerativni učinek na tkivu. Uporablja se kot samostojna ali spremljajoča terapija.

http://www.votan.si/svetlobna-terapija

Odpri sliko ➜

Odpri sliko ➜

Treating Depression With Light

Here is good news for depressed people! You can often make yourself happier simply by lighting up your life! With some advice from your doctor, you can relieve depressive symptoms by getting more bright light.

Just as darkness makes us sad, bright light tends to restore us to a normal level of cheer. This simple and very helpful principle helps us treat depression.

You can help depression by spending more time outdoors in daylight, but to be frank,
changing habits to experience more daylight is often not practical in today’s world.

That is why we usually recommend increased artificial lighting. This section will explain how bright light treatment is used, but if you have significant depression, you should check with your doctor.

3.A. Dosage of light
People vary in how much bright light they need to combat depressive symptoms. There must be many factors, including how depressed a person is. A modest increase in lighting may help with a mild problem.

A more severe problem may need much brighter lighting and a longer duration of bright lighting to overcome. The amounts of light needed should at least bring a depressed person above the average for daily light exposure.

This can be achieved with as little as 30 minutes of very bright light near 10,000 lux (like sunshine) or with a couple hours of light of about 2500 lux (like a cloudy day or deep shade).

If the amount of depression is substantial, light much dimmer than 2,000 lux may not be very effective without many hours of daily exposure, though in the chapter on advanced sleep phase, I will describe how modest amounts of lighting may be sufficient for certain sleep problems. We have to admit that the information doctors now have about the effectiveness of different lighting dosages is quite fragmentary. We really cannot give very exact advice about what brightness and duration of light a particular person should use. (Incidentally, doctors have a similar problem in recommending the dosage needed of antidepressant drugs or the amount of psychotherapy needed.) Thus, for the present time, somebody who wants to use bright light might need to try to see empirically how much light is needed.

Using light treatment for up to several hours a day might be beneficial at the beginning of treating serious depressions. Most people would be able to decrease the duration of treatment after a satisfactory response was obtained. Initial durations of 30 min. for 10,000 lux or 2 hours for 2–3,000 lux are often satisfactory.[17]

I would be uncertain of the safety of increasing the brightness of light treatment above 10,000 lux, so I never recommend any brighter treatment.

The decision about whether to try 10,000 lux for a shorter time or 2,000–3,000 lux for a somewhat longer time depends on several considerations. Most people prefer the brighter light for the shorter time because of the convenience of shortening the time required. However, using 10,000 lux does generally require getting close to the light source, which may be awkward in some settings where it is convenient to use a lower intensity for a longer time. A full 10,000 lux will also make some people’s eyes uncomfortable or increase headache and eyestrain. Theoretically, we would expect 2,000 lux to be safer for the eyes than 10,000 lux, but 10,000 lux has been tested enough now without causing any eye damage that most experts seem quite confident of its safety. After all, 10,000 is no brighter than what we normally experience when we spend time outdoors on a bright day. People have been testing 10,000 lux for longer than recorded history. I will describe just how to provide this bright light in another section below.

Like many other habits – eating, exercising, and sleeping, for example – a person may need to try different amounts of bright light to discover how much is best individually. If a half hour a day is not doing enough after a couple of weeks, try one hour or two hours. If 2000 lux is not doing enough, try 10,000 lux. Although some people will experience some mood improvement within an hour of a single dose of bright light, it often takes a week of daily bright light treatment for a severely depressed person to feel a measurable improvement, and increasing benefit may be felt as treatment continues for at least 8 weeks. Unfortunately, we do not have adequate controlled trials of treatment longer than 8 weeks, but that is true of many antidepressant drugs as well. It is likely that continuing improvement or prevention of relapse will continue for many months. One should not become disappointed too quickly, especially if even slow improvement is seen. On the other hand, it is possible to use too much bright light. The dosage in time or brightness sometimes needs to be adjusted and reduced. Eyestrain, headache, irritability, and sleep problems may be signs of excessive treatment. In a later section, we will consider some specific side effects on sleep and mood which may require reduction of dosages.

3.B. The time of day to use bright light
For many people, bright light at any time of day will help depression. It appears that most people will get the best benefit from bright light very early in the morning, even starting an hour before the usual time of awakening. However, there is a smaller group who seem to benefit more with evening light. I think it is the sleep pattern which provides the most useful clinical clue to optimal timing.

The principles for optimizing light timing from sleep patterns are simple. If a person tends to have trouble falling asleep and has trouble getting up on time each morning, that person is likely to do best with using bright light early in the morning. People whose depression is linked to sleeping more may also tend to do best with light in the morning. For such people, using bright light immediately after awakening is the most effective time to use bright light. Indeed, some studies suggest that getting up a bit early to use morning bright light increases its benefit. Dr. Terman recommends beginning morning light treatment about 2 ½ hours after the middle of sleep.[18] For example, if a person sleeps from midnight to 8 AM (with midsleep at 4 AM), the recommended time would be 6:30 AM. In contrast, evening bright light may be best for the person who tends to nod off early in the evening, who cannot stay awake for prime time television, and who awakens earlier than desired in the morning. For the person who does not fit clearly into either of these patterns, there might be little difference between morning and evening bright light treatment, but morning light would be the better bet. We will explore these principles in more detail in the section on advanced and delayed sleep phase syndromes.

For the person suited for morning light, a way to get fast relief is to use wake therapy. The way to use wake therapy is to get up in the middle of sleep, turn on bright room lights, and stay awake for the second half of the night (e.g., a person who sleeps from midnight to 8 AM would get up at 4 AM). Then, bright light treatment is commenced near the normal awakening time. Most depressed people experience surprising relief of depression after getting up so early, provided they stay awake all day despite some increased sleepiness. Although patients who are not using bright light tend to relapse after wake therapy as soon as they sleep again, with bright light treatment, the rapid gains of wake therapy are often maintained. I believe that a single night of wake therapy (getting up in the middle of the night) is helpful and practical at the beginning of light treatment, and getting up a bit early thereafter might help.

A special case are the patients with bipolar disorder (a history of being manic depressive), especially those whose mood cycles rapidly between mania and depression. These patients may be prone to trigger unwanted and serious mania with use of bright light, especially in the morning, or with wake therapy. Mid-afternoon bright light may be the best for these patients, according to preliminary testing. I do not recommend wake therapy for bipolar patients, because of the risk of mania, unless they are in a doctor's active care.

Please see below, in Chapter 7, the warning against manic-depressives using light or wake therapy without a mood stabilizer.



3.C. Choices of lighting
The most important aspect of treatment lighting is that it be bright. So far as we know, sunlight and artificial lighting work equally well. However, in various climates, environments, and social situations, using sunlight may be impractical, so most people will need to buy, rent, or borrow special artificial lighting. So far as we know, both ceiling lighting that hits the eyes and lighting level with the eyes work equally. There is some indication – not yet proven clinically – that light coming from below (e.g., a light on the floor) would not work as well.

So far as we know, diffuse incandescent and fluorescent light of equal brightness might work equally, but there are two important advantages with fluorescent fixtures. First, since fluorescent lighting is more energy-efficient, you pay for less electricity and produce less wasted heat with fluorescents. That heat could be annoying in the summer. Second, fluorescent tubes are large, so it is easy to diffuse the light over an area of several square feet. This means that unlike the very bright point of light produced by incandescent bulbs, fluorescents produce somewhat dimmer light over a larger area. When the light goes through the lens of the eye and hits the retina (the back of the eye), the energy of diffuse light is spread over a large area, so it should not heat the retina or make you see spots (like the spots you see after looking at a flash bulb). Diffused bright light is safer for the eyes and will cause less strain. As a general rule, you could not burn your retina by staring at any of the common fluorescent bulbs with diffusers, even if you are receiving 10,000 lux.
Obviously, nobody should stare at a light if it hurts or seems to dim the vision. Some people are annoyed by the flicker or sound of older fluorescent ballasts, so fluorescent fixtures with electronic (high frequency) ballasts will probably cause less headache and sense of eye strain. Avoid the older models flickering with the 60 cycles household alternating current.

I am not certain that staring at very bright incandescent bulbs is entirely within the range of safety. I would never recommend that anyone take any unnecessary chances with their eyes, so I feel that nobody should treat themselves by looking directly at bright incandescent light (such as 300 watt halogen bulbs) without diffusers. Indirect incandescent lighting or lights with large diffusers or shades should be safe, as I will discuss in the section about using lighting for the elderly. The problem with using indirect incandescent lighting for treating depression is that the standard commercial lamps lose most of the brightness by bouncing the light off the walls and ceiling, partly because the light travels a greater distance. As a result, the lighting store “torchiere” incandescent lamps are probably not bright enough to do the job well for serious depressions. Another problem is that the 300–500 watt halogen incandescent bulbs used commonly do not last very long and may be a bit difficult to replace. For these reasons, all of the lighting fixtures which I can currently recommend for significant depression use fluorescent bulbs and diffusers.

Recently, there have been technologic developments with LED lighting. LEDs are more energy-efficient than even fluorescent bulbs, and they will last longer. However, except for a few studies with the “Litebook” brand LED devices, which have been shown to have beneficial effects as compared to placebo, there has been relatively little testing of the white LED devices. Some of the products such as Litebook models use LEDs to produce intense white light from a rather small area, which might produce more glare or discomfort. I am not aware of sufficient testing to make me as confident of these LED treatment devices as the fluorescent designs. The truth is, there have been virtually no controlled comparisons available between different models and brands.

New research has shown that the “photometer” cells in the retina of the eye which respond to brightness (the intrinsically photosensitive retinal ganglion cells) are most sensitive to blue and blue-green light. These cells contain a special visual pigment called melanopsin which responds best to blue. On the other hand, there also seems to be a benefit in additional colors of light, such as some green mixed with the blue. The issue is complicated by the fact that the lenses in our eyes turn yellow as we age, so that blue light does not reach the retina as well as green or yellow in older people. There have been several studies which suggest that blue LED light of only moderate intensity will influence the circadian system more than white light of the same intensity. The hope is that blue light which seems less bright and requires less electricity could be as useful as brighter white light. However, this advantage of blue light may not hold in middle-aged and elderly people. Moreover, I know of no evidence that blue light works better than white light for treating problems with mood. Although the blue LED devices are thought to be safe, their margin of safety may not be as great as with white light, because animal studies show that blue light can be much more damaging to the retina. All in all, as of 2012, I am inclined to feel more confidence with the white fluorescent models.

There are now a large and confusing number of light treatment models and manufacturers advertised on the internet. Several manufacturers (listed in the box below) make fluorescent fixtures which do a very nice job of helping depressed patients. A good place to find lighting manufacturers is at the website for the Society for Light Treatment and Biological Rhythms.[19] In general, 160–300 watts of fluorescent light illuminating a bright diffuser about 1 yard from the eyes will give about 2,000–3,000 lux. The exact brightness depends on various aspects such as the bulbs, the diffusers, and the reflectors. To get 10,000 lux, the manufacturers may recommend a somewhat bigger fixture with more wattage or placing the fixture closer to the eyes, perhaps 12" to 18". There is considerable difference in the brightness of different models, so the dimmer models may need to be closer to the eyes. Some of the designs hold the light tilted a bit above the eyes, which seems to be convenient for getting light treatment and also getting something done reading, writing, eating or watching TV.

Some researchers have recommended that people stare at the fluorescent diffuser most of the time when they are getting the treatment. Others have recommended glancing at the light every minute or two. Others seem to feel that having the light source anywhere in the field of vision (even if you aren’t looking at it directly) is just as good. Unfortunately, we really do not know yet whether it makes much difference whether you look directly at the lighting. Most evidence suggests that having the light within the field of vision and glancing at it occasionally is sufficient. Also, unfortunately, we really do not know if it makes a difference where the light is during treatment: above the eyes (tilted), straight in front, to the side, or even below where the person is looking. There is some evidence – not fully convincing – that light slightly above or even with the eyes would be more effective than light coming from below where the eyes are looking.

I wish we had some sort of independent testing of different brands and models of bright lights,
to see which ones helped depressed people the most. We do not. Comparative testing would be difficult and expensive, so almost none has been done. I suppose that all of the fluorescent light boxes quoted to give the same brightness are likely
to be roughly equivalent, but I do not really know.

A lot has been written about natural lighting and whether one should use lighting with a “full” spectrum. I suspect this spectrum of claims is largely baloney! In fact, the FDA forced one company advertising “full spectrum” light into a consent decree admitting that their claims were deceptive.[20] First of all, almost any white light produces the full visible spectrum of colors (light wave lengths). The question is the balance of the different wavelengths, which does differ from one light source to another. If one looks at the fine spectrum with a precision spectrophotometer (which measures the color balance of light exactly), I doubt that any of the commercial sources really produces a light spectrum which could be mistaken for the rather smooth spectrum of natural sunlight. Fortunately, the eye is not a spectrophotometer, and there is no evidence that the sunlight spectrum is necessary.

The main issue is how much ultraviolet light the light source produces, because some of the “full spectrum”
bulbs give off enough ultraviolet to possibly increase cataracts or skin cancer. There is no evidence that the ultraviolet is needed for the bright light treatment benefit, so needless to say, I do not recommend anything with significant ultraviolet.

Most of the fluorescent manufacturers use a plastic diffuser which filters out the harmful ultraviolet. There is more discussion of the risks of ultraviolet in the chapter about risks. It is true that many Americans get so little ultraviolet sunlight in the winter that they become vitamin D deficient, especially when fear of obesity unwisely persuades people to avoid Vitamin-D-fortified dairy products. It is sun striking the skin, not the eyes, which helps with Vitamin D,
so if you are going to a tanning saloon for Vitamin D, keep the ultraviolet out of your eyes.
Then light up your eyes without the ultraviolet. Anyone concerned about vitamin D would probably be wiser to buy some in the grocery store than to try ultraviolet lighting to prevent vitamin deficiency. Dairy products can be good for you. Did you know that you might actually get depressed, if your cholesterol is too low?

There has been a good deal of hoopla and advertising about light visors.
The idea is that if you could wear the light source on your head like a baseball cap – and get bright light treatment without blinding yourself – it might be easier to go about your business. I never recommend light visors, because I am not the least bit convinced that they work for treating depression. In fact, there have been several studies suggesting that various light visors which have been tested do not work better than ineffective dummy treatments.

Apparently a more promising approach to making light treatment easy is providing light exposure during sleep. There is some evidence that gradually increasing light toward the end of the night, simulating dawn, has a useful effect.

[21] I do not think that the dawn simulation idea has yet been proven superior to using a steady intensity of light during sleep. There has not yet been sufficient research on this technique from different laboratories. I do not recommend trying dawn simulation unless it is quite impossible to arrange for sufficient light when you are awake, but some manufacturers provide dawn simulation devices, if you want to take a chance on trying light before waking up. Light before waking up is a form of morning light, and it probably will work best for people with trouble falling asleep and with trouble getting up on time.

http://www.brightenyourlife.info/ch3.htmlhttp://www.brightenyourlife.info/ch3.html

Svetlobna terapija proti depresiji
piše Jana Polajnar
Svetloba in tema vplivata na življenjski ritem in človeške aktivnosti. Ljudje smo "narejeni" za naraven ritem od zore do mraka, noč pa je za počitek. Žleze in hormoni delajo v svojem ritmu, kar pa ni v skladu s sodobnim načinom življenja in tako prihaja do težav in na koncu lahko tudi do obolenj. Prav za depresijo in težave s težo je značilno, da simptomi dosežejo svoj vrhunec ponoči in pozimi. Tako velja razmisliti, da bi si namesto tablet, privoščili malo sončka! Danes so na voljo svetila BIOPTRON (sončna svetloba z odvzetim UV sevanjem), ki nam to omogočajo. Terapija je naravna in spodbuja telo k samozdravljenju.

1. Svetloba in barve proti stresu in strahu

Bližajo se dolgi in temačni večeri, ko se nas rado poloti malodušje, na dan prilezejo različni strahovi in že čutimo, da se nas "loteva" depresija. Zakaj bi jo čakali, da bo prišla kot vedno, ko smo sami? Pojdimo v svet svetlobe in barv!
Dokazano je, da sončna svetloba in barvitost v našem okolju povzročijo in ohranjajo dobro voljo in dobro notranje počutje.
Barvna in svetlobna terapija sta dopolnilni terapiji in dokazano krepita spanec, lajšata stres in krepko pomagajo pri premagovanju strahu.

2. Kronična utrujenost

Svetlobna in barvna terapija sta odlična dopolnilna in alternativna metoda in pomoč pri kronični utrujenosti, izčrpanosti, izgorelosti, pri pomanjkanju volje, motnjah v čustvovanju, pri napadih panike ali depresije, težavah s koncentracijo ipd.

Istočasno z razpoloženjskimi motnjami pogosto pride do oslabitve imunskega sistema, kardiovaskularnih težav, itd. itd., zdravje se nam začne hitro krhati in težavam ne vidimo konca.

3. Barvna in svetlobna terapija - kaj je to in kako deluje?

S svetlobno in barvno terapijo zagotavljamo ravnovesje in krepitev življenjske energije, ki nam ohranja fizično zdravje ter harmonijo telesa, uma in duha.

Dokazano odlično pomaga pri

celjenju ran (po operacijah, poškodbah, preležanine, razjede itd.);

lajšanju bolečin(revmatologija, fizioterapija itd.);

kožnih obolenjih (luskavica, akne);

boleznih otrok in novorojenčkov (vnetja dihal, kostnomišične poškodbe);

sezonskih razpoloženjskih motnjah, zmanjšani koncentraciji, kronični utrujenosti itd.

podpira odpornost,

povečuje energijske rezerve (slaba koncentracija in slab spomin),

pozitivno vpliva na presnovo in prebavo,

krepi spanec,

lajša stres itd.

Fiziki pravijo, da je barva vidna energija z določeno frekvenco. Ko barvna energija pride v naše telo, sproži različne fiziološke procese. Tako razumemo, zakaj barve vplivajo na naše misli, razpoloženje in obnašanje! Barve pa lahko vplivajo tudi na določene organe in posledično pomagajo pri preprečevanju, zdravljenju nekaterih bolezni in pri rehabilitaciji.
http://www.neganog.com/Svetlobna_terapija_proti_depresiji.php

Ah, škoda truda, res, kaj sploh sprašujem. Ti, Saphire, preprosto nič ne veš. Pa ni problem, povej, da ne veš, pa je. Saj jaz tudi ne vem kaj je svetlobna terapija. Guglat pa znam, veš, me ni treba tu zasipat s kilometri tekstov. Sem mislila, da boš v dveh, treh stavkih sama povedala.

Odpri sliko ➜

Ce znas guglati, potem ves, prav tako kot vem jaz 🙂))
Ce te tema moti, jo preskoci in zapri.
Simples. 🙂 😆